By: Marc Sorenson, Sunlight Institute–
In 1997, Dr. Y Sato and colleagues showed that sunlight deprivation (due to being hospitalized) in Parkinson’s patients resulted in compensatory hyperparathyroidism, which in turn led to reduced bone mass and excessive hip fractures.[1] In 1998, he made the same observation regarding elderly women with Alzheimer’s disease.[2] Then, in 2003, he reported that sunlight deprivation was also a cause of hip fractures in elderly women who suffered from stroke.[3] This time, however, he studied the effects of sunlight—or the lack thereof—on the bone mass of elderly women who were either exposed to sunlight or were kept inside a care facility. Over twelve months, 129 women were exposed to regular sunlight and another 129 received no sunlight exposure. The results were startling: in these sedentary women, the sunlight group increased bone mass by an average 3.1%; in the non-sunlight-exposed group, it decreased by 3.3%, a swing of 6.4%.
Of course, one might ask why a small increase in bone density in one group and a loss of bone density in the other makes any difference; the real question is whether it prevented hip fractures. Now consider this: as proof of the efficacy of improving bone mass, the women who had the benefit of sunlight had only one bone fracture in their group. The sunlight-deprived group had six fractures! This is obviously a reversal of osteoporosis and a reversal of fracture risk.
Sato was not through with his research; in 2005, he and his colleagues exposed a group of Alzheimer’s patients to sunlight for one year, and another was kept in a typical indoor hospital setting.[4] In the sunlight group a 220% increase in vitamin D levels was found, and bone mass increased by 2.7%. In the indoor group, bone mass decreased by 5.6%. That is a difference of 7.3% in only one year! The final proof, of course, is with fractures. In the sunlight group, there were three fractures; in the sunlight-deprived group there were eleven, or 3.7 times more.
Finally, in 2011, Dr. Sato and his group did a similar study on elderly patients with Parkinson’s disease.[5] A two-year program of sunlight exposure was compared to a two-year program of continued sunlight deprivation. This time, the sunlight group experienced an increase of 3.8% bone mass, whereas the sunlight-deprived group lost 2.6% bone mass. The sunlight group experienced three fractures and the sunlight deprived group, eleven fractures, as in the aforementioned study.
One more thought: An investigation in Spain concluded that women who actively participated in sun exposure had one-eleventh the chance of a hip fracture as those who stayed indoors.[6]
The conclusion: sunlight exposure not only prevents, but can also reverse osteoporosis and fractures. Every physician who treats this disease should have sunlight exposure as his number-one treatment protocol. But is anyone listening?
[1] Sato Y, Kikuyama M, Oizumi K. High prevalence of vitamin D deficiency and reduced bone mass in Parkinson’s disease. Neurology. 1997 Nov;49(5):1273-8.<?xml:namespace prefix = o />
[2] Sato Y, Asoh T, Oizumi K. High prevalence of vitamin D deficiency and reduced bone mass in elderly women with Alzheimer’s disease. Bone. 1998 Dec;23(6):555-7.
[3] Sato, Y. Amelioration of osteoporosis and hypovitaminosis D by sunlight exposure in stroke patients. Neurology 2003;61:338-42.
[4] Sato Y, Iwamoto J, Kanoko T, Satoh K. Amelioration of osteoporosis and hypovitaminosis d by sunlight exposure in hospitalized, elderly women with Alzheimer’s disease: a randomized controlled trial. J Bone Miner Res. 2005;20:1327-33.
[5] Sato Y, Iwamoto J, Honda Y. Amelioration of osteoporosis and hypovitaminosis D by sunlight exposure in Parkinson’s disease. Parkinsonism Relat Disord 2011;17(1):22-6.
[6] Larrosa, M. Vitamin D deficiency and related factors in patients with osteoporotic hip fracture. Med Clin (BARC) 2008;130:6-9.
